Show simple item record

dc.contributor.authorLiao, Xiaofeng
dc.contributor.authorPirapakaran, Tharshikha
dc.contributor.authorLuo, Xin M.
dc.date.accessioned2017-09-18T09:35:37Z
dc.date.available2017-09-18T09:35:37Z
dc.date.issued2016-06-14
dc.identifier.citationXiaofeng Liao, Tharshikha Pirapakaran, and Xin M. Luo, “Chemokines and Chemokine Receptors in the Development of Lupus Nephritis,” Mediators of Inflammation, vol. 2016, Article ID 6012715, 15 pages, 2016. doi:10.1155/2016/6012715
dc.identifier.urihttp://hdl.handle.net/10919/78933
dc.description.abstractLupus nephritis (LN) is a major cause of morbidity and mortality in the patients with systemic lupus erythematosus (SLE), an autoimmune disease with damage to multiple organs. Leukocyte recruitment into the inflamed kidney is a critical step to promote LN progression, and the chemokine/chemokine receptor system is necessary for leukocyte recruitment. In this review, we summarize recent studies on the roles of chemokines and chemokine receptors in the development of LN and discuss the potential and hurdles of developing novel, chemokine-based drugs to treat LN.
dc.format.mimetypeapplication/pdf
dc.language.isoenen_US
dc.publisherHindawien_US
dc.rightsCreative Commons Attribution 4.0 International
dc.rights.urihttps://creativecommons.org/licenses/by/4.0/
dc.titleChemokines and Chemokine Receptors in the Development of Lupus Nephritisen_US
dc.typeArticle - Refereed
dc.date.updated2017-09-18T09:35:37Z
dc.description.versionPeer Reviewed
dc.rights.holderCopyright © 2016 Xiaofeng Liao et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
dc.title.serialMediators of Inflammationen_US
dc.identifier.doihttps://doi.org/10.1155/2016/6012715
dc.type.dcmitypeText


Files in this item

Thumbnail
Thumbnail

This item appears in the following Collection(s)

Show simple item record

Creative Commons Attribution 4.0 International
License: Creative Commons Attribution 4.0 International